The regulatory mechanism of HR cell death via MARK1
Project/Area Number |
15K07319
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Research Category |
Grant-in-Aid for Scientific Research (C)
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Allocation Type | Multi-year Fund |
Section | 一般 |
Research Field |
Plant protection science
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Research Institution | Okayama University |
Principal Investigator |
Matsui Hidenori 岡山大学, 環境生命科学研究科, 助教 (20598833)
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Project Period (FY) |
2015-04-01 – 2018-03-31
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Project Status |
Completed (Fiscal Year 2017)
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Budget Amount *help |
¥4,940,000 (Direct Cost: ¥3,800,000、Indirect Cost: ¥1,140,000)
Fiscal Year 2017: ¥1,560,000 (Direct Cost: ¥1,200,000、Indirect Cost: ¥360,000)
Fiscal Year 2016: ¥1,690,000 (Direct Cost: ¥1,300,000、Indirect Cost: ¥390,000)
Fiscal Year 2015: ¥1,690,000 (Direct Cost: ¥1,300,000、Indirect Cost: ¥390,000)
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Keywords | リン酸化 / 植物免疫 / 細胞死 / RNA metabolism / リン酸化プロテオミクス / R-gene / エフェクター / Rタンパク質 / 耐病性 / 過敏感細胞死 / タンパク質相互作用 |
Outline of Final Research Achievements |
To reveal the plant immune components, we have performed phosphoproteomics approach, and identified PSIG1 that functions as a negative regulator of cell death during pathogen infection. For exploring of the molecular function of PSIG1, we assessed the subcellular localization of PSIG1, and identified PSIG1 interacting protein. One of the interactors of SMG7 which functions as a regulator of mRNA decay in Arabidopsis thaliana. PSIG1 physically binds to SMG7, and PSIG1 and SMG7 were co-localized around P-bodies using Agroinfiltration method in Nicotiana benthamiana. These results suggest that PSIG1 might regulate mRNA metabolism for controlling cell death during pathogen infection.
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Report
(4 results)
Research Products
(20 results)
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[Journal Article] Chitin nanofiber elucidates the elicitor activity of polymeric chitin in plants2015
Author(s)
Egusa, M., Matsui, H., Urakami, T., Okuda, S., Ifuku, S., Nakagami, H. and Kaminaka, H.
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Journal Title
frontiers in Plant Science
Volume: 6
Pages: 1098-1098
DOI
Related Report
Peer Reviewed / Open Access / Int'l Joint Research / Acknowledgement Compliant
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