| Budget Amount *help |
¥16,380,000 (Direct Cost: ¥12,600,000、Indirect Cost: ¥3,780,000)
Fiscal Year 2020: ¥2,470,000 (Direct Cost: ¥1,900,000、Indirect Cost: ¥570,000)
Fiscal Year 2019: ¥4,160,000 (Direct Cost: ¥3,200,000、Indirect Cost: ¥960,000)
Fiscal Year 2018: ¥4,160,000 (Direct Cost: ¥3,200,000、Indirect Cost: ¥960,000)
Fiscal Year 2017: ¥5,590,000 (Direct Cost: ¥4,300,000、Indirect Cost: ¥1,290,000)
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| Outline of Final Research Achievements |
Nitrogen is a major limiting nutrient in plant growth and productivity. In flooded paddy soils, wetland rice, an important mainstay crop, preferentially takes up ammonium as an abundant inorganic nitrogen source. However, an ammonium supply at high concentration is frequently toxic to many terrestrial plants. Therefore, ammonium influx into plant roots via the high-affinity transport system (HATS) is down-modulated under elevated external ammonium. HATS-responsible, plasma membrane-located ammonium transporter 1 (AMT1) proteins are inactivated via phosphorylation of the conserved threonine residue at the cytoplasmic carboxyl-tail under elevated external ammonium. We identified the role of a protein kinase, ACTPK1, in phosphorylation and inactivation of ammonium-induced AMT1;2 in ammonium-preferring rice grown under sufficient ammonium. This finding will help to improve nitrogen use efficiency in rice and confer ammonium-resistance on ammonium-sensitive plants.
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