Functional analyses of fad104 on survival just after birth and insulin sensitivity.
| Project/Area Number |
20790070
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| Research Category |
Grant-in-Aid for Young Scientists (B)
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| Allocation Type | Single-year Grants |
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| Research Field |
Biological pharmacy
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| Research Institution | Nagoya City University |
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Principal Investigator |
NISHIZUKA Makoto Nagoya City University, 大学院・薬学研究科, 講師 (00363953)
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| Project Period (FY) |
2008 – 2009
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| Project Status |
Completed (Fiscal Year 2009)
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| Budget Amount *help |
¥4,290,000 (Direct Cost: ¥3,300,000、Indirect Cost: ¥990,000)
Fiscal Year 2009: ¥2,080,000 (Direct Cost: ¥1,600,000、Indirect Cost: ¥480,000)
Fiscal Year 2008: ¥2,210,000 (Direct Cost: ¥1,700,000、Indirect Cost: ¥510,000)
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| Keywords | 肥満 / 糖尿病 / 脂肪細胞分化 / 新生児疾患 / 遺伝子発現 / 肺形成 / fad104 / 新生児 / ノックアウトマウス / サーファクタント / インスリン感受性 |
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| Research Abstract |
In this study, we demonstrated that fad104, a positive regulator of adipogenesis, has a crucial role for regulating the formation of white adipose tissue and insulin sensitivity. Furthermore, we also demonstrated that fad104 is essential for lung development just after birth.
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Report
(3 results)
Research Products
(25 results)
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[Journal Article] Disruption of the novel gene fad104 causes rapid postnatal death and attenuation of cell proliferation, adhesion2009
Author(s)
Nishizuka M., Kishimoto K., Kato A., Ikawa, M., Okabe, M., Sato R., Niida H., Nakanishi M., Osada S., Imagawa M.
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Journal Title
spreading and migration Exp. Cell Res. 315(5)
Pages: 809-819
Related Report
Peer Reviewed
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[Journal Article] Disruption of the novel gene fadl04 causes rapid postnatal death and attenuation of cell proliferation, adhesion, spreading and migration2009
Author(s)
Nishizuka M., Kishimoto K., Kato A., Ikawa M., Okabe M., Sato R., Niida H., Nakanishi M., Osada S. and Imagawa M.
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Journal Title
Experimental Cell Research 315(5)
Pages: 809-819
Related Report
Peer Reviewed
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