| Project/Area Number |
22370019
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| Research Category |
Grant-in-Aid for Scientific Research (B)
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| Allocation Type | Single-year Grants |
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| Section | 一般 |
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| Research Field |
Plant molecular biology/Plant physiology
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| Research Institution | Nara Institute of Science and Technology |
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Principal Investigator |
TASAKA Masao 奈良先端科学技術大学院大学, バイオサイエンス研究科, 教授 (90179680)
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| Co-Investigator(Kenkyū-buntansha) |
UCHIDA Naoyuki 奈良先端科学技術大学院大学, バイオサイエンス研究科, 助教 (40467692)
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| Project Period (FY) |
2010 – 2012
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| Project Status |
Completed (Fiscal Year 2012)
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| Budget Amount *help |
¥18,460,000 (Direct Cost: ¥14,200,000、Indirect Cost: ¥4,260,000)
Fiscal Year 2012: ¥6,240,000 (Direct Cost: ¥4,800,000、Indirect Cost: ¥1,440,000)
Fiscal Year 2011: ¥6,240,000 (Direct Cost: ¥4,800,000、Indirect Cost: ¥1,440,000)
Fiscal Year 2010: ¥5,980,000 (Direct Cost: ¥4,600,000、Indirect Cost: ¥1,380,000)
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| Keywords | R タンパク質 / UNI / レセプターキナーゼ / ERECTA / リガンド / EPFLL / Rタンパク質 / EPFL / 次世代シークエンサー / SNP |
|---|
| Research Abstract |
The constitutive active R-protein, uni-1D, induced abnormal morphogenesis in Arabidopsis shoot. We have isolated suppressors of uni-1D. One of them encodes RPT2, which is a component of 26S proteasome and binds directly to UNI. Erecta, which is a receptor kinase in a plasma membrane, is also one of the suppressors. ER family genes expressed outside of SAM, however, affect the SAM activity. ER expressed in phloem affect the inflorescence stem elongation and the EPFL4 and 6, both of which expressed in endoderm cells, function as a ligand in this process.
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