| Project/Area Number |
25860267
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| Research Category |
Grant-in-Aid for Young Scientists (B)
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| Allocation Type | Multi-year Fund |
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| Research Field |
Human pathology
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| Research Institution | Gifu University |
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Principal Investigator |
GOTO Naoe 岐阜大学, 医学(系)研究科(研究院), 非常勤講師 (80444280)
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| Project Period (FY) |
2013-04-01 – 2015-03-31
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| Project Status |
Completed (Fiscal Year 2014)
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| Budget Amount *help |
¥2,080,000 (Direct Cost: ¥1,600,000、Indirect Cost: ¥480,000)
Fiscal Year 2014: ¥1,170,000 (Direct Cost: ¥900,000、Indirect Cost: ¥270,000)
Fiscal Year 2013: ¥910,000 (Direct Cost: ¥700,000、Indirect Cost: ¥210,000)
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| Keywords | 濾胞性リンパ腫 / クロマチン再構成 / クロマチン再構成因子複合体 / ARID1A / ElonginA / 十二指腸濾胞性リンパ腫 |
|---|
| Outline of Final Research Achievements |
We found that ARID1A expression was downregulated in 42 of 80 follicular lymphoma specimens, in contrast, ARID1A was lost in 11 of 16 duodenal follicular lymphoma.
This result suggested that molecular mechanism or etiology of duodenal follicular lymphoma would be much different to that to nodal follicular lymphoma.
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