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2017 Fiscal Year Final Research Report

The regulatory mechanism of HR cell death via MARK1

Research Project

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Project/Area Number 15K07319
Research Category

Grant-in-Aid for Scientific Research (C)

Allocation TypeMulti-year Fund
Section一般
Research Field Plant protection science
Research InstitutionOkayama University

Principal Investigator

Matsui Hidenori  岡山大学, 環境生命科学研究科, 助教 (20598833)

Project Period (FY) 2015-04-01 – 2018-03-31
Keywordsリン酸化 / 植物免疫 / 細胞死 / RNA metabolism
Outline of Final Research Achievements

To reveal the plant immune components, we have performed phosphoproteomics approach, and identified PSIG1 that functions as a negative regulator of cell death during pathogen infection. For exploring of the molecular function of PSIG1, we assessed the subcellular localization of PSIG1, and identified PSIG1 interacting protein. One of the interactors of SMG7 which functions as a regulator of mRNA decay in Arabidopsis thaliana. PSIG1 physically binds to SMG7, and PSIG1 and SMG7 were co-localized around P-bodies using Agroinfiltration method in Nicotiana benthamiana. These results suggest that PSIG1 might regulate mRNA metabolism for controlling cell death during pathogen infection.

Free Research Field

植物病理学

URL: 

Published: 2019-03-29  

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