2017 Fiscal Year Final Research Report
The regulatory mechanism of HR cell death via MARK1
Project/Area Number |
15K07319
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Research Category |
Grant-in-Aid for Scientific Research (C)
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Allocation Type | Multi-year Fund |
Section | 一般 |
Research Field |
Plant protection science
|
Research Institution | Okayama University |
Principal Investigator |
Matsui Hidenori 岡山大学, 環境生命科学研究科, 助教 (20598833)
|
Project Period (FY) |
2015-04-01 – 2018-03-31
|
Keywords | リン酸化 / 植物免疫 / 細胞死 / RNA metabolism |
Outline of Final Research Achievements |
To reveal the plant immune components, we have performed phosphoproteomics approach, and identified PSIG1 that functions as a negative regulator of cell death during pathogen infection. For exploring of the molecular function of PSIG1, we assessed the subcellular localization of PSIG1, and identified PSIG1 interacting protein. One of the interactors of SMG7 which functions as a regulator of mRNA decay in Arabidopsis thaliana. PSIG1 physically binds to SMG7, and PSIG1 and SMG7 were co-localized around P-bodies using Agroinfiltration method in Nicotiana benthamiana. These results suggest that PSIG1 might regulate mRNA metabolism for controlling cell death during pathogen infection.
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Free Research Field |
植物病理学
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