2012 Fiscal Year Final Research Report
Investigation of new signal transduction and cell function of ADAM-type hemorrhagic toxin
| Project/Area Number |
22590061
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| Research Category |
Grant-in-Aid for Scientific Research (C)
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| Allocation Type | Single-year Grants |
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| Section | 一般 |
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| Research Field |
Biological pharmacy
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| Research Institution | Nagoya University |
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Principal Investigator |
ARAKI Satohiko 名古屋大学, 大学院・理学研究科, 講師 (80242808)
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| Co-Investigator(Kenkyū-buntansha) |
SAWADA Hitoshi 名古屋大学, 大学院・理学研究科, 教授 (60158946)
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| Project Period (FY) |
2010 – 2012
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| Keywords | 細胞生物学 / 毒素 |
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| Research Abstract |
ADAM-type hemorrhagic toxins induce cell fragmentation on vascular endothelial cells. A signaling molecule was identified as the responsible factor regulating the cell fragmentation, and shown to perform on various cell types. It suggests one of regulating mechanism of cell morphology is clarified.
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Research Products
(8 results)
