Identification of positional signals regulating shoot epidermal cell fate

2014 Fiscal Year Final Research Report

• Project/Area Number: 22687003
• Research Category: Grant-in-Aid for Young Scientists (A)
• Allocation Type: Single-year Grants
• Research Field: Plant molecular biology/Plant physiology
• Research Institution: Osaka University
• Principal Investigator: TAKADA Shinobu 大阪大学, 理学(系)研究科(研究院), 助教 (40456992)
• Research Collaborator: TAKADA Nozomi ; YOSHIDA Ayaka ; IIDA Hiroyuki ; ITO Miharu
• Project Period (FY): 2010-04-01 – 2015-03-31
• Keywords: 細胞分化 / 位置情報 / パターン形成 / 胚発生 / 転写制御 / シロイヌナズナ

Outline of Final Research Achievements

Cell-type specific transcription factors play key roles in determining cell fate through the regulation of gene expression. However, molecular mechanisms regulating the cell-type dependent activity of transcriptional factors are largely unknown. Our studies showed that ATML1 functions as a master transcriptional regulator for shoot epidermal cell fate in Arabidopsis thaliana. We developed new tools to identify key components that restrict ATML1 expression to the outermost cell layer.

Free Research Field

植物発生生物学