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2013 Fiscal Year Final Research Report

Analysis of DNA repair, cell cycle progression and cell death in dedifferentiation and regeration of renal tubular cells

Research Project

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Project/Area Number 23591194
Research Category

Grant-in-Aid for Scientific Research (C)

Allocation TypeMulti-year Fund
Section一般
Research Field Kidney internal medicine
Research InstitutionKochi University

Principal Investigator

HIRANO SEIKI  高知大学, 教育研究部医療学系, 助教 (20368616)

Co-Investigator(Kenkyū-buntansha) TERADA Yoshio  高知大学, 教育研究部医療学系, 教授 (30251531)
Project Period (FY) 2011 – 2013
Keywords腎尿細管障害
Research Abstract

The number of hemodialysis patient is increasing, which is a critical problem in Japan. To analyze repair and regeneration of renal tubular cells after renal failure will contribute the way of preventive therapy of renal disease. I found that renal tubular cells express the markers of de-differentiation after cell damage and they form tubule when the marker is expressed. I am trying to do further analyses.

  • Research Products

    (1 results)

All 2011

All Presentation (1 results)

  • [Presentation] Six2-GDNF Pathway Is Activated during Experimental Acute Kidney Injury and Plays a Crucial Role in Renal Tubular Regeneration2011

    • Author(s)
      Seiki Hirano, et al.
    • Organizer
      American Society of Nephrology Kidney Week2011
    • Place of Presentation
      ペンシルベニアコンベンションセンター、フィラデルフィア,USA
    • Year and Date
      2011-11-12

URL: 

Published: 2015-07-16  

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