Is cytoplasmic male sterility explained by mitochondrial overactivation?

2015 Fiscal Year Final Research Report

• Project/Area Number: 25292001
• Research Category: Grant-in-Aid for Scientific Research (B)
• Allocation Type: Partial Multi-year Fund
• Section: 一般
• Research Field: Science in genetics and breeding
• Research Institution: Hokkaido University
• Principal Investigator: Kubo Tomohiko 北海道大学, (連合)農学研究科(研究院), 教授 (40261333)
• Co-Investigator(Kenkyū-buntansha): TAGUCHI Kazunori 農業・食品産業技術総合研究機構, 北海道農業研究センター, 主任研究員 (80414754) ; KURODA Yosuke 農業・食品産業技術総合研究機構, 北海道農業研究センター, 研究員 (40595071)
• Project Period (FY): 2013-04-01 – 2016-03-31
• Keywords: ミトコンドリア / 細胞質雄性不稔性

Outline of Final Research Achievements

Cytoplasmic male sterility (CMS) is a breeding character that makes hybrid seed production feasible. In the genetic model of CMS, male sterility expresses when the cytoplasm has male-sterility inducing factor while the nucleus has two recessive alleles of restorer-of-fertility gene (rf). The cytoplasmic factor is a mitochondrial gene that is specific to CMS mitochondrial genome. Such genes have been found from many plant species but the mechanism how CMS expresses is still obscure. The objective of this study is to clarify the mitochondrial function of CMS sugar beet. We found the followings: biochemical difference between male-fertile and CMS mitochondria; male-sterility induction by F0F1-ATPase alteration; and effect of novel Rf on the male-sterility inducing mitochondrial gene.

Free Research Field

分子育種学