2017 Fiscal Year Final Research Report
tRNA intron-degrading protein complex regulating plant mitochondrial disease
| Project/Area Number |
26252007
|
|---|
| Research Category |
Grant-in-Aid for Scientific Research (A)
|
| Allocation Type | Single-year Grants |
|---|
| Section | 一般 |
|---|
| Research Field |
Plant protection science
|
|---|
| Research Institution | Kagawa University |
|---|
Principal Investigator |
|
|---|
| Project Period (FY) |
2014-04-01 – 2018-03-31
|
| Keywords | 宿主特異的毒素 |
|---|
| Outline of Final Research Achievements |
Seven proteins located in mitochondria were selected from 13 candidate proteins from AmBP30 protein complex responsible for mRNA processing of ACR-toxin receptor gene ACRS. AmBP30 protein which binds to ACRS mRNA, was localized in mitochondria only from ACR-toxin insensitive citrus cultivars. ACRTS1, ACRTS2, ACRTS3, and ACRTS4 were isolated as cluster genes for ACT-toxin biosynthesis enzymes and their functions were analyzed. Immunoprecipitation using anti-ACTT1 antibodies identified all 9 enzymes which had been known to have a responsibility for ACT-toxin biosynthesis, as well as additional new 7 candidate enzymes and their genes for the toxin biosynthesis.
|
| Free Research Field |
農学
|
