| Project Area | Plant regulatory systems that control developmental interactions between meristems and lateral organs |
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| Project/Area Number |
19060003
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| Research Category |
Grant-in-Aid for Scientific Research on Priority Areas
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| Allocation Type | Single-year Grants |
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| Review Section |
Biological Sciences
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| Research Institution | Nagoya University |
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Principal Investigator |
MACHIDA Yasunori 名古屋大学, 大学院・理学研究科, 特任教授 (80175596)
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| Co-Investigator(Kenkyū-buntansha) |
MACHIDA Chiyoko 中部大学, 応用生物学部, 教授 (70314060)
TAKAHASHI Hiro 千葉大学, 園芸学研究科, 准教授 (30454367)
IWASAKI Yukimoto 福井県立大学, 生物資源学部, 教授 (20193732)
SASABE Michiko 弘前大学, 農学生命科学部, 准教授 (00454380)
吉岡 泰 名古屋大学, 大学院・理学研究科, 准教授 (60202397)
上野 宜久 名古屋大学, 大学院・理学研究科, 助教 (20335011)
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| Co-Investigator(Renkei-kenkyūsha) |
YOSHIOKA Yasushi 名古屋大学, 大学院・理学研究科, 准教授 (60202397)
UENO Yoshihisa 名古屋大学, 大学院・理学研究科, 助教 (20335011)
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| Project Period (FY) |
2007 – 2012
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| Project Status |
Completed (Fiscal Year 2012)
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| Budget Amount *help |
¥204,700,000 (Direct Cost: ¥204,700,000)
Fiscal Year 2012: ¥34,500,000 (Direct Cost: ¥34,500,000)
Fiscal Year 2011: ¥34,500,000 (Direct Cost: ¥34,500,000)
Fiscal Year 2010: ¥34,500,000 (Direct Cost: ¥34,500,000)
Fiscal Year 2009: ¥34,500,000 (Direct Cost: ¥34,500,000)
Fiscal Year 2008: ¥34,500,000 (Direct Cost: ¥34,500,000)
Fiscal Year 2007: ¥32,200,000 (Direct Cost: ¥32,200,000)
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| Keywords | 葉の発生分化 / miRNA / 遺伝子発現制御 / 葉の左右相称性 / 葉の表裏 / DNAのメチル化 / 細胞分裂周期 / 葉の向軸面・背軸面の発生会化 / ETT/ARF3, ARF4発現制御 / TGS / PTGS / DNAメチル化 / サイトカイニン合成酵素遺伝子 |
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| Research Abstract |
Arabidopsis ASYMMETRIC LEAVES2 (AS2) and AS1 genes control leaf morphogenesis from the upstream of its developmental pathway, but the regulatory framework for the morphogenesis remained to be demonstrated. Our group has investigated a role of these genes in adaxial-abaxial establishment and formation of symmetric flat leaves. We have demonstrated that AS2 and AS1 proteins form a complex, which plays a key role for the development of leaves with such characteristic architectures by repressing expression of abaxial gene ETTIN/ARF3 in presumptive adaxial domain in leaf primordia. The repression mechanism includes systems including binding of the complex to the ETTIN promoter region, enhancement of the level of miR390 that is involved in degradation of ETTIN mRNA, and epigenetic silencing of ETTIN.
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