| Project/Area Number |
22770044
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| Research Category |
Grant-in-Aid for Young Scientists (B)
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| Allocation Type | Single-year Grants |
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| Research Field |
Plant molecular biology/Plant physiology
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| Research Institution | Kumamoto University |
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Principal Investigator |
TAKECHI Katsuaki 熊本大学, 大学院・自然科学研究科, 准教授 (70515501)
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| Co-Investigator(Renkei-kenkyūsha) |
TAKANO Hiroyoshi 熊本大学, バイオエレクトリクス研究センター, 教授 (70242104)
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| Project Period (FY) |
2010 – 2011
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| Project Status |
Completed (Fiscal Year 2011)
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| Budget Amount *help |
¥4,160,000 (Direct Cost: ¥3,200,000、Indirect Cost: ¥960,000)
Fiscal Year 2011: ¥1,430,000 (Direct Cost: ¥1,100,000、Indirect Cost: ¥330,000)
Fiscal Year 2010: ¥2,730,000 (Direct Cost: ¥2,100,000、Indirect Cost: ¥630,000)
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| Keywords | オルガネラ / 細胞壁 / コケ植物 / 葉緑体 / 斑入り / 細胞死(PCD) |
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| Research Abstract |
Bacterial LrgB gene related to control system for peptidoglycan degradation is widely conserved in plant genome and plant LrgB genes are thought to derived from cyanobacteria which is believed as ancestor of plastid. We examined the physiological role of LrgB in angiosperm Arabidopsis and moss Physcomitrella. In Arabidopsis, AtLrgB functions against new cell death control system in which chloroplast participates, similar to the bacterial Lrg sytem, whereas Physcomitrella PpLrgB is not found to function against cell death. Although there are the differences of phenotype between both LrgB genes disrupted lines, expression of AtLrgB gene in the Physcomitrella PpLrgB disrupted line complemented the phenotype of it. We will examine whether the function of LrgB has changed along the plant evolution.
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