| Project/Area Number |
24659125
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| Research Category |
Grant-in-Aid for Challenging Exploratory Research
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| Allocation Type | Multi-year Fund |
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| Research Field |
General medical chemistry
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| Research Institution | Gunma University |
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Principal Investigator |
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| Co-Investigator(Kenkyū-buntansha) |
UCHIYAMA Tsuyoshi 群馬大学, 生体調節研究所, 研究員 (40420103)
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| Project Period (FY) |
2012-04-01 – 2015-03-31
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| Project Status |
Completed (Fiscal Year 2014)
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| Budget Amount *help |
¥2,080,000 (Direct Cost: ¥1,600,000、Indirect Cost: ¥480,000)
Fiscal Year 2014: ¥520,000 (Direct Cost: ¥400,000、Indirect Cost: ¥120,000)
Fiscal Year 2013: ¥650,000 (Direct Cost: ¥500,000、Indirect Cost: ¥150,000)
Fiscal Year 2012: ¥910,000 (Direct Cost: ¥700,000、Indirect Cost: ¥210,000)
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| Keywords | 糖尿病 / メタボリックシンドローム / 脂肪細胞 / 肥満 / インスリン分泌 / 膵臓疲弊 / 高血圧 |
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| Outline of Final Research Achievements |
We can not significantly express constitute active heat shock factor1. However, we have the information about metabolic syndrome bellow. The hypertensive agents reduced lipoprotein lipase expression in the mature adipocyte, but in immature adipocyte, the hypertensive agents induced lipoprotein lipase expression. we accepted the journal PLOS one at 17. September. The title is iAngiotensin II reduces lipoprotein lipase expression in visceral adipose tissue via phospholipase C β4 depending on feeding but increases lipoprotein lipase expression in subcutaneous adipose tissue via c-Src,
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