2016 Fiscal Year Final Research Report
Mechanism analysis, suppresion of EAON by DC Transfected with PU.1.
| Project/Area Number |
26861471
|
|---|
| Research Category |
Grant-in-Aid for Young Scientists (B)
|
| Allocation Type | Multi-year Fund |
|---|
| Research Field |
Ophthalmology
|
|---|
| Research Institution | Tokyo Medical University |
|---|
Principal Investigator |
|
|---|
| Research Collaborator |
Umazume Akihiko 東京医科大学, 医学部, 助教 (20617568)
Yamakawa Naoyuki 東京医科大学, 医学部, 助教 (80599134)
|
|---|
| Project Period (FY) |
2014-04-01 – 2017-03-31
|
| Keywords | PU.1 / EAE / EAON |
|---|
| Outline of Final Research Achievements |
In this study, we investigated PU.1 expression in the retina of mice with experimental autoimmune uveoretinitis (EAU) and the association between PU.1 expression level and inflammation in EAU.
PU.1 may play crucial roles in the development and progression of inflammation in EAU
|
| Free Research Field |
遺伝治療 神経眼科 ぶどう膜炎
|
